New facts are coming to light about him death of 9-year-old Georginaas according to toxicological control Other anesthetics besides ketamine were reportedly detected in her body.
Specifically, according to information published, during the inspection carried out at the Forensic Medicine Service of Athens, as in addition to benzodiazepines and ketamine, anesthetics were found. lidocaine and prilocaine.
According to the same information, the specifics substances have been put under the microscope of the authorities and a quantitative control is expected for the dosage of each substance.
These substances were not included in the medication that little Georgina was taking from the Children’s Hospital and she may have given them together with the ketamine. her mother. According to the document of the toxicological tests, the benzodiazepines found are Midazolam, nordazepam, clobazam and norclobazam, while the anesthetics contain the substances ketamine, norketamine, lidocaine and prilocaine.
The 9-year-old was also given antiepileptic drugs, phenobarbital, levetiracetam and valproic acid.
The paper from the hospital with the medicines given to her by the nurses shows otherwise.
It turns out that the doctors did not administer anesthetics to the child. Experts estimate that a toxic cocktail had been created in the child’s body, which shows the magnitude of the horror.
The medical examiner Mr. Tsantiris speaking in SKAI reported that the ketamine found in Georgina’s body was in an overdose and was certainly not given by the doctors.
With regard to the substances which are now being investigated, it is noted that:
THE lidocaine (lidocaine) is a local anesthetic and belongs to the category of aminoamides. It causes reversible blockade of the transmission of impulses along the central and peripheral nerve pathways. Their action is believed to be due to inhibition of the passage of sodium ions in cell membranes resulting in slowing of depolarization to a degree that does not develop energy potential.
THE prilocaine (prilocaine) binds to the intracellular surface of sodium channels and blocks the subsequent influx of sodium into the cell, thereby limiting the transmission of electrical stimuli. The block is reversible and when prilocaine diffuses away from the cell, sodium channel function is restored, as is the smooth transmission of nerve impulses.
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